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1
CREM deficiency in mice alters the response of bone to intermittent parathyroid hormone treatment
CREM deficiency in mice alters the response of bone to intermittent parathyroid hormone treatment
Liu, F., | Elsevier Inc | 2007 ,
2
Osteopenia in transgenic mice with osteoblast-targeted expression of the inducible cAMP early repressor
Osteopenia in transgenic mice with osteoblast-targeted expression of the inducible cAMP early repressor
Chandhoke Taranpreet K,, | Elsevier Inc | 2008 ,
3
Col3.6-HSD2 transgenic mice: A glucocorticoid loss-of-function model spanning early and late osteoblast differentiation
Col3.6-HSD2 transgenic mice: A glucocorticoid loss-of-function model spanning early and late osteoblast differentiation
Yang, M., | Elsevier Inc | 2010 ,
4
Selective osteoblast overexpression of IGF-I in mice prevents low protein-induced deterioration of bone strength and material level properties
Selective osteoblast overexpression of IGF-I in mice prevents low protein-induced deterioration of bone strength and material level properties
Brennan-Speranza, T. C, | Elsevier Inc | 2011 ,
5
New roles and mechanism of action of BMP4 in postnatal tooth cytodifferentiation
New roles and mechanism of action of BMP4 in postnatal tooth cytodifferentiation
Gluhak-Heinrich, J., | Elsevier Inc | 2010 ,
6
Effects of global or targeted deletion of the EP4 receptor on the response of osteoblasts to prostaglandin in vitro and on bone histomorphometry in aged mice
Effects of global or targeted deletion of the EP4 receptor on the response of osteoblasts to prostaglandin in vitro and on bone histomorphometry in aged mice
Gao, Q., | Elsevier Inc | 2009 ,
7
Transgenic mice with osteoblast-targeted insulin-like growth factor-I show increased bone remodeling
Transgenic mice with osteoblast-targeted insulin-like growth factor-I show increased bone remodeling
Jin Jiang., | Elsevier Inc | 2006 ,
8
Postnatal growth and bone mass in mice with IGF-I haploinsufficiency
Postnatal growth and bone mass in mice with IGF-I haploinsufficiency
Jianing He., | Elsevier Inc | 2006 ,
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